Inhibition of Murine AIDS (MAIDS) Development in C57BL/6J Mice by Tyrphostin AG-1387
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چکیده
منابع مشابه
Tyrphostin AG 126 inhibits development of postoperative ileus induced by surgical manipulation of murine colon.
Manipulation of the bowel during abdominal surgery leads to a period of ileus, which is most severely manifested after procedures that directly involve the colon. Ileus is associated with the increased expression of proinflammatory cytokines and chemokines, a leukocytic infiltration into the muscularis, and the release of mediators from resident and infiltrating leukocytes that directly inhibit...
متن کاملDisruption of microtubules in living cells by tyrphostin AG-1714.
Tyrphostin AG-1714 and several related molecules with the general structure of nitro-benzene malononitrile (BMN) disrupt microtubules in a large variety of cultured cells. This process can be inhibited by the stabilization of microtubules with taxol or by pretreatment of the cells with pervanadate, which inhibits tyrosine phosphatases and increases the overall levels of phosphotyrosine in cells...
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15 صفحه اولTyrphostin AG126 Inhibits the Development of Postoperative Ileus Induced by Surgical Manipulation of the Murine Colon
Background: Manipulation of the bowel during abdominal surgery leads to a period of ileus, which is most severely manifested after procedures that directly involve the colon. Ileus is associated with the increased expression of pro-inflammatory cytokines and chemokines, a leukocytic infiltration into the muscularis, and the release of mediators from resident and infiltrating leukocytes that dir...
متن کاملCD4+ T cells are required for development of a murine retrovirus- induced immunodeficiency syndrome (MAIDS)
Mice depleted in vivo of CD4+ Th cells by treatment with mAb GK1.5 were found to be resistant to the lymphoproliferative/immunodeficiency disease (MAIDS) induced in intact mice by infection with the mixture of LP-BM5 murine leukemia viruses. Depleted mice did not develop lymphadenopathy or splenomegaly, had normal serum IgM levels, normal CTL responses to alloantigens, and were able to generate...
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ژورنال
عنوان ژورنال: Virology
سال: 2000
ISSN: 0042-6822
DOI: 10.1006/viro.2000.0642